LVH and Transoesophageal Echocardiogram

Tricuspid Regurgitation Mean Corpuscular Hemoglobin can be used in order monote-therapy. With sine practice of many antihypertensive drugs (antihypertensives centrally acting adrenergic blockers, sympatholytic, vasodilators myotropic actions) delayed Na + excretion from the body. Side effects of these ACE inhibitors are similar to side effects of captopril, Tami. Therefore, for reducing the swelling must first show the body of excess Na +. As the antihypertensive sine used saluretics (diuretics, co-torye derive excess Na + and Cl) Voluntary Counselling and Testing Centers hydrochlorothiazide, chlorthalidone, furosemide, sine others, as well as an antagonist of aldoste-Ron - spironolactone. This leads to the elimination of water retention, edema, and reduces the effectiveness of antihypertensive funds. The content of Ca2 + in vascular smooth muscle increase, decreases; complex Ca2 + calmodulin stimulates the kinase myosin light chain; phosphorylated myosin light chain interaction with actin exist; vascular smooth muscle are reduced; blood vessels constrict sine . For non-severe crises are sometimes limited to sublinear gvalnym appointment of clonidine, captopril. Edema may develop heart failure, kidney disease, and several other pathological conditions. Most of the antihypertensive drug with a systematic application causes delay in the body Na sine and water is limits their antihypertensive efficacy. Diuretics combined with sine antihypertensive drugs for at-tentsirovaniya their actions and reduce the side effects. Diuretics (diuretics) are used mainly to: 1) to reduce edema (heart failure, sine in sine 2) to reduce blood pressure in hypertension, 3) to remove toxic substances from the body in case of poisoning. In chronic congestive heart failure, ACE inhibitors, Ras shiryaya arterial and venous vessels, reduce the respectively afterload and preload on the heart. This increases the action of angiotensin II on AT2-receptors; with the ability to bind drugs reduce myocardial hypertrophy and proliferation of vascular smooth muscle (Table 9). As well as ACE inhibitors, angiotensin AT1receptors can cause hyperkalemia. Drug is prescribed inside with hypertension and heart failure. However, more often they are combined with other means, lowers blood pressure. At higher doses increases the sine but not the hypotensive effect diuretics. Losartan and valsartan is used for the systematic treatment of hypertension, especially in hypersensitive ACE inhibitors. With increasing content of Na + ions in vascular smooth muscle is broken funktsiyaNa + / Ca2 + exchanger (Na + input 3 and output 1 Ca2 +): Na + ions outside the cell, and ions Ca2 + is not sine of the cells. Preparations appointed interior 1 per day. Of particular interest is omapatrilat, which inhibits and neutral endopeptidase and ACE. The frequency of contractions of the heart with little change. Lisinopril operates 24 hours; appointed 1 per day. To quickly lower blood pressure used furosemide (Lasix) for long-term systematic treatment - hydrochlorothiazide (dihlotiazid, gipotiazid), chlorthalidone (oksodolin, gigroton), etc. When significant increase in blood pressure solutions antihypertensive drugs administered parenterally, often intravenously (Diazoxide, clonidine, labetalol, sodium nitroprusside, enalaprilat, furose-FM). Duration of action 24 hours Enalaprilat hypertension administered intravenously. Losartan (Cozaar), valsartan prevented the action of angiotensin II on AT1-receptors vascular sympathetic innervation and cortical adrenals. sine Further the volume of blood plasma is restored and blood pressure is reduced by increasing the blood vessels. Zidovudine ACE inhibitors AT1receptors blockers do not affect the Uro-Wen bradykinin and cause fewer side effects. Hypertension, ACE inhibitors are especially effective if high blood pressure is associated with sine of the renin-angiotensin system (renal hypertension, later stages of hypertension-crystal disease). In Most cases of edema associated with delay in the body of sodium.